pesta re vurona.
pesta re vurona.
ksydia
πως ακριβως τρωγεται? ολοκληρα καρυδια? και όταν λες γινεται τσαι, πως γινεται τσαι αν μιλαμε για ολοκληρα καρυδια? και για τι ποσοτητα μιλάμε?Originally Posted by Raven Lord
χεχχεχεχ
1ον πρέπει να πάρεις τα ολόκληρα καρύδια και όχι το τριμμένο και
20ν και σημαντικότερο, ΔΕΝ ΚΑΠΝΙΖΕΤΑΙ! γίνεται τσάι ή τρώγεται αλλά όχι σε τσιγάρο γιατί η θερμοκρασία του καταστρέφει τη "δραστική" ουσία.
so this is permanence
poutses.
mple.
i salvia den entassetai stin commonly known ntrogka edw ki afou fytrwnei sto mexico 8a tan ligaki diskolo na iparxei nomothesia giautin (paradoksws bebaia iparxei nomo8esia gia to 5-meo-dmt stin ellada (check erowid)).
bebaia an 8imaste ena (oxi para poly) prosfato reportage tou star, eixan katasxe8ei megales posotites salvias ("periergo eksotiko parais8isiogono fyto"(?~!?)) stin ellada.
http://www.erowid.org/plants/nutmeg/nutmeg_info2.shtml
Rock or Nothing
ότι θα πει ο τσοπντ.
After the Triumph of Your Birth.
ayto me to mosxokarydo to eixa kanei alliws. to ebala sto mylo tou kafe kai to etripsa kai efaga ayto. se ftiaxnei ligo, alla den mporw na fantastw poso prepei na fas gia na deis parais8hseis. h nomimh odos den exei na proteinei kati kalytero omws
h mpanana kanei ontws douleia an exeis thn ypomonh na kaneis kamia 5 tsigara, alla bebaia 8a peis "den epina enan garo na teleiwnoume?" egw to ebrasa poly ligo kai epiase pantws. de nomizw na sou dinei parais8hseis, einai polu elafry
to entelws narkwtiko einai ta fylla ortansias alla mhn to kanete. einai dhlhthriwdh kai epikindyna
tsoflia apo fystikia aiginhs den exw dokimasei proswpika alla mou lene oti den kanei douleia ki alloi. parepiptontws otan kati exei asxhmh geysh to anakateyeis me menta gia na to kapniseis
an pas se giatro kai peis oti koimasai olh thn hmera paizei na sou grapsei amfetamines, afou se gamhsei stis e3etaseis, opote mallon prepei na exeis ontws problhma gia na ton peiseis.
ena gnwsto alla asxhmo ftia3imo einai to pontikofarmako. to opoio to anakateyoun kai aytoi pou sou poulane me alla narkwtika gia oikonomia. enas filos leei kai gia katsaridoktono alla exei keraies kai den ton empisteyomai
exei dokimasei kaneis na sfa3ei fryno, na ton gdarei, na ton 3eranei kai na kapnisei to derma tou?
mia fora eixa kapnisei rigani tiligmeni se fillo apo tetradio me grammes k eixa sterewsei to tsigaro me seloteip
Cut your flesh and worship Santa
Elege tipota?
Σιγά μη βάλεις να ακούει ο Ling Ling κλαψό-το-παιδί-πονάει-πονάει-ο-Αρτέμης-μουνιές και αρχίσεις να τον γλύφεις.exei dokimasei kaneis na sfa3ei fryno, na ton gdarei, na ton 3eranei kai na kapnisei to derma tou?
Ελάχιστοι ως κανένας παίζει να το καταλάβουν, αλλά αφού δεν έχετε γούστο στη μουσική, στα καρτούνς θα έχετε;
Αν πιστευες οτι δε θα το πιαναμε το υπονοουμενο, τοτε παιζει να εισαι τοσο gay οσο ο Xandir.
They say we've got a lifetime, but we know that ain't true.
Strychnine (pronounced /strɪkniːn/ (British) or /strɪknaɪn/ (U.S.)) is a very toxic (LD50 = 1 mg/kg), colourless crystalline alkaloid used as a pesticide, particularly for killing small vertebrates such as rodents. Strychnine causes muscular convulsions and eventually asphyxia or sheer exhaustion. The most common source is from the seeds of the Strychnos nux-vomica tree. Strychnine is the most bitter substance in the world. Its taste is detectable in concentrations as low as 1 ppm.
Strychnine acts as a blocker or antagonist at the inhibitory or strychnine-sensitive Glycine receptor (GlyR), a ligand-gated chloride channel in the spinal cord and the brain.
Formula - C21H22N2O2
LD50 - 1 mg/kg
Molecular mass - 334.41 g/mol
CAS number - 57-24-9
SMILES - [H][C@]([C@@](C(C=CC=C7)=C7N34)5[C@H]6N(CC5)C2)3 [C@@]1([H])[C@@H](C6)[C@] 2=CCO[C@H]1CC4=O
Strychnine poisoning in humans
Strychnine poisoning can be fatal to humans, by inhalation, swallowing or absorption through eyes or mouth. It produces some of the most dramatic, terrifying, best known, and painful symptoms imaginable. For this reason, strychnine poisoning is often used in literature and film.
Ten to twenty minutes after exposure, every muscle in the body will start to simultaneously contract, starting with the head and neck. The spasms then spread to every muscle in the body, with nearly continuous convulsions. They get worse at the slightest stimulus. They progress, increasing in intensity and frequency until the backbone arches continually. Death comes from asphyxiation caused by paralysis of the brain's breathing apparatus, or by exhaustion from the convulsions. At that time, the body "freezes," even in the middle of a convulsion. Rigor mortis sets in immediately, with the eyes left wide open.
Treatment involves giving depressants, such as diazepam (intravenously), to control the convulsions, and giving an activated charcoal infusion to drink, serving to absorb any poison remaining within the digestive system. If the patient lives 24 hours, recovery is probable.
Strychnine poisoning in animals
Strychnine poisoning in animals occurs usually from ingestion of baits designed for use against rodents (especially gophers and moles) and coyotes. Rodents baits are commonly available over-the-counter, but coyote baits are illegal in the United States. However, since 1990 in the United States most baits containing strychnine have been replaced with zinc phosphide baits.[1] The most common domestic animal to be affected is the dog, either through accidental ingestion or intentional poisoning. An approxiamite lethal dose for a dog is 0.75 mg per kg body weight.[2] For a 0.3% strychnine bait, five grams could be enough to kill a twenty kilogram dog.
The onset of symptoms is 10 to 120 minutes after ingestion.[2] Symptoms include seizures, a "sawhorse" stance, and opisthotonus (rigid extension of all four limbs). Death is usually secondary to respiratory paralysis. Treatment is by detoxification using activated charcoal, pentobarbital for the symptoms, and artificial respiration for apnea.
Strychnine in drugs
There is a common but wrong urban legend that strychnine is added to drugs like LSD or that strychnine is present in the peyote cactus or in psilocybin mushrooms. It should be noted that the dose of LSD is so small that it could not be mixed with a toxic amount of strychnine, even if strychnine made up an entire blotter square.
(On the issue of strychnine in LSD:
The following text was written by Alexander T. Shulgin in response to the overwhelming misconception that strychnine is commonly found in street samples of LSD:
"The observation of strychnine as being present in any street drug, as a by-product, or a contaminant, or an impurity has never been documented. It is a natural plant product, as are the ergots which are used in the synthesis of LSD. But they come from totally unrelated plants; there has never been a report of strychnine and an ergot alkaloid co-existing in a single species. So if the two materials are together in a drug sample, it could only be by the hand of man. I have personally looked a large number of illicit street offerings and have never detected the presence of strychnine. The few times that I have indeed found it present, have been in legal exhibits where it usually occurred in admixture with brucine (also from the plant Strychnos nux-vomica) in criminal cases involving attempted or successful poisoning.
The same argument applies to the myth that occasionally surfaces, that strychnine occurs in the white tufts of peyote. This is equally fraudulent -- it has never been reported in that cactus or any other cactus."
Furthermore, it should probably be spelled out that strychnine is not needed to bond LSD to blotter paper, nor is strychnine a breakdown product of LSD. these are probably the two most commonly repeated gross misconceptions.
The source of the "strychnine is commonly found in LSD" myth may be somewhat grounded in truth. For example, in "LSD: My Problem Child" Albert Hofmann cites a case in the late sixties of Strychnine being found in an "LSD" sample that was a white powder. However, what is commonly claimed is that strychnine is found in a significant percentage of LSD, specifically blotter LSD, which is *not* true. Shulgin's note that he has analyzed many samples of LSD and never found strychnine is backed up by published analyses done by PharmChem and the LA County Street Drug Analysis program, which likewise never found any strychnine.
This is intuitively backed up by the fact that a 5mm x 5mm "standard" square of blotter LSD only weights about 2mg and if the paper itself was made completely out of pure strychnine it is still on the very low end of Strychnine's threshold of activity.
Strychnine is not the cause of tracers, cramps, nausea, or amphetamine-like LSD-effects. Its possible that poorly synthesized LSD might have other ergot derivatives in it, which might contribute to the harsh body load that some get on taking LSD. Also, the very close chemical relatives 1-Methyl-LSD and 1-Acetyl-LSD (which break down into LSD in aqueous solution) might be present in some street samples and might contribute to the harsh body load. (Petter Stafford has claimed in his _Psychedelics Encyclopedia_ that 1-Acetyl-LSD is supposedly "smoother" than d-LSD -- thus "strychnine laced acid" may acutally be pure d-LSD, while "pure lsd" may be 1-Acetyl-LSD or some substitute). And the chemicals iso-LSD and lumi-LSD which are breakdown products of LSD might contribute to the body loading on some trips, particularly via a hypothetical synergistic effect. Given this plethora of possible chemicals in street "LSD", its not needed to look to a chemical which has hardly ever been found in analyzed samples to explain variations in the strength and "cleanliness" of street acid.
Its also possible that LSD itself simply causes adverse physical effects, particularly muscle cramping, in persons suceptible to it. The reported side effects of LSD (the nausea and apparent CNS stimulant effects) are commonly reported side effects of seritonergic drugs such as fluoxetine (Prozac) and buspirone (Buspar), and also are commonly reported (and typically more severe) with other psychedelics like Mescaline.
Or its quite likely that the "strychnine" reactions to LSD are entirely psychosomatic. Both Leary ("The Psychedelic Experience") and Lilly ("Programming and Metaprogramming...", "Center of the Cyclone") have each observed this reaction in people who cannot handle the surge of emotion associated with a trip.
Further advice would be to avoid methylxanthines (caffiene, theophylline in tea, etc) prior to dosing. Some have noted a possible synergistic effect between them and LSD causing, or contributing, to a harsh body load during a trip. And prior use of dramamine may alleviate the nausea sometimes associated with LSD, and other psychedelic drugs (although it may also effect the quality of the trip -- Shulgin has noted in PiHKAL that he shuns the use of anti-nauseants in order to experience the effects of the psychedelic, both good and bad, with no possible interference).
In summary, it can't be said that we know specifically why sometimes acid feels "cleaner" than other times. However, based on the availability of plausible explanations, and the evidence of drug analysis, and general implausiblity of the whole strychnine concept, we can conclude that it isn't due to any concentration of strychnine. Also, while it can't completely be ruled out, the presence of strychnine in LSD is so minimal that the majority of LSD users will never once come across it.)
Some folks like water
Some folks like wine
But I like the taste
Of straight strychnine (hey, hey)
You may think it's funny
That I like this stuff
But once you've tried it
You can't get enough (wow!)
Wine is red
Poison is blue
Strychnine is good
For what's ailin' you
If you listen to what I say
You'll try strychnine some day
Make you jump, it'll make you shout
It'll even knock you out
Strychnine hey, hey
Πόσο κομματάρα.
never tell
O rockavlon to egrapse?
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